Что бы это значило
Модератор: Pyankov Vasily
Что бы это значило
Что бы это значило:
- Вложения
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- 1) NH3 (perfusion agent)
2) FDG (metabolic agent) before glucose load
3) FDG after glucose load - CARDIAC PET SHORT AXIS.JPG (147.37 КБ) 11560 просмотров
- 1) NH3 (perfusion agent)
-
- 1) NH3 (perfusion agent)
2) FDG (metabolic agent) before glucose load
3) FDG after glucose load - CARDIAC PET HLA.JPG (145.24 КБ) 11558 просмотров
- 1) NH3 (perfusion agent)
-
- 1) NH3 (perfusion agent)
2) FDG (metabolic agent) before glucose load
3) FDG after glucose load - CARDIAC PET VLA.JPG (150.43 КБ) 11584 просмотра
- 1) NH3 (perfusion agent)
- Кто на ком стоял?... потрудитесь излагать ваши мысли яснее.(c)
Последний раз редактировалось Ren_Yumi Чт июн 14, 2007 9:55 pm, всего редактировалось 1 раз.
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- Site Admin
- Сообщения: 3780
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Ян, а ты бы не могла прокоментировать результаты исследования? Если я правильно понимаю, что перфузия и метаболизм не нарушены, то следует остановиться на состояниях, сопровождающихся изолированной дилатацией правого желудочка.
Это может быть:
легочная гипертензия (первичная, ТЭЛА, ХОБЛ),
пороки (трикуспидальная недостаточность, обструкция выносящего тракта ПЖ, стеноз клапана легочной артерии, надклапанный стеноз легочной артерии),
болезни правого желудочка (аритмогенная дисплазия правого желудочка, пергаментный правый желудочек).
Хотелось бы посмотреть Эхо и ЭКГ.
Это может быть:
легочная гипертензия (первичная, ТЭЛА, ХОБЛ),
пороки (трикуспидальная недостаточность, обструкция выносящего тракта ПЖ, стеноз клапана легочной артерии, надклапанный стеноз легочной артерии),
болезни правого желудочка (аритмогенная дисплазия правого желудочка, пергаментный правый желудочек).
Хотелось бы посмотреть Эхо и ЭКГ.
Сейчас посмотрю, есть ли другие исследования.
Последний раз редактировалось Ren_Yumi Чт июн 14, 2007 11:18 pm, всего редактировалось 2 раза.
EXAM: CARDIAC MRI
PROCEDURE DATE: 04/19/2007
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INTERPRETATION: History: 38 year-old woman with syncope.
Examination: Magnetic resonance examination of the heart was performed using gradient echo and double inversion recovery acquisition sequences. In addition, perfusion and delayed hyper enhancement imaging was obtained as well.
Observations:
The main pulmonary artery, and both left and right pulmonary arteries are moderately dilated. In addition, there is intraluminal signal indicating turbulent flow. These findings are consistent with increased pulmonary resistance and pulmonary hypertension. Right ventricular free wall myocardium appears thickened. In addition, there is clockwise rotation of the heart flattening of the interventricular septum. These findings consistent with a right ventricular pressure and volume loading and right ventricular dilatation and hypertrophy. No evidence of tricuspid or mitral valve dysfunction is noted.
Cine examination shows thinning of the interventricular septum throughout the cardiac cycle and mild hypokinesia both right and left ventricles. However, no regional wall motion abnormality is noted.
After intravenous administration of contrast, perfusion imaging was obtained. No region of right or left ventricular myocardium fail to enhance.
10 minutes after intravenous administration of contrast, delayed hyper enhancement imaging was obtained. No region of right or left ventricular myocardium enhanced.
IMPRESSION: Evidence of pulmonary hypertension and right ventricular dysfunction as described above. However, there is no evidence of delayed hyper enhancement arguing against infiltration of the myocardium with sarcoidosis.
PROCEDURE DATE: 04/19/2007
.
INTERPRETATION: History: 38 year-old woman with syncope.
Examination: Magnetic resonance examination of the heart was performed using gradient echo and double inversion recovery acquisition sequences. In addition, perfusion and delayed hyper enhancement imaging was obtained as well.
Observations:
The main pulmonary artery, and both left and right pulmonary arteries are moderately dilated. In addition, there is intraluminal signal indicating turbulent flow. These findings are consistent with increased pulmonary resistance and pulmonary hypertension. Right ventricular free wall myocardium appears thickened. In addition, there is clockwise rotation of the heart flattening of the interventricular septum. These findings consistent with a right ventricular pressure and volume loading and right ventricular dilatation and hypertrophy. No evidence of tricuspid or mitral valve dysfunction is noted.
Cine examination shows thinning of the interventricular septum throughout the cardiac cycle and mild hypokinesia both right and left ventricles. However, no regional wall motion abnormality is noted.
After intravenous administration of contrast, perfusion imaging was obtained. No region of right or left ventricular myocardium fail to enhance.
10 minutes after intravenous administration of contrast, delayed hyper enhancement imaging was obtained. No region of right or left ventricular myocardium enhanced.
IMPRESSION: Evidence of pulmonary hypertension and right ventricular dysfunction as described above. However, there is no evidence of delayed hyper enhancement arguing against infiltration of the myocardium with sarcoidosis.