Ребенок с Precocious puberty получает декапептил I.M. Года три уже получает. Сегодня пришли за очередным рецептом за 5 минут до конца смены и заодно рассказали ( мама рассказала) что ребенок ( ему 14) много пьет и много писает. Сахар крови в норме. (Когда я болела, они, оказывается, были с этой проблемой у сменного врача, и сахар крови он им сделал).
Ждать ли мне неприятностей от декапептила? И, вообще, что делать по этому поводу?
Precocious puberty
Модераторы: Ren_Yumi, Alon, dr.Ira
Precocious puberty
Делай, что должен, и будь, что будет.
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Melnichenko
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Igor Bulatov
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- Зарегистрирован: Сб апр 21, 2007 3:31 am
DIABETES INSIPIDUS
Approximately 90% of vasopressin secretion needs to be interrupted before there is clinical diabetes insipidus. The commonest tumors causing diabetes insipidus in childhood are craniopharyngioma and germinoma. The latter are often occult for many years and patients require serial neuro-imaging before the diagnosis may be revealed. It may be as long as 21 years from the onset of diabetes insipidus to imaging the tumor (22). In a child who has diabetes insipidus, it is important to monitor carefully growth and screen for an evolving anterior pituitary endocrinopathy.
Lesions in the pituitary stalk associated with the presence of diabetes insipidus, are usually situated in the middle of the stalk and related to a dural inflammatory process, such as sarcoidosis or Langerhans cell histiocytosis (LCH) (23). Langerhans cell histiocytosis is not a cancer, but has similar characteristics and is a clonal proliferation of abnormal histiocytes, the Langerhans cells (24). A common presentation of LCH in the middle childhood years, which used to be described as Hand-Schüller-Christian disease, are small punched-out skull lesions associated with pituitary stalk thickening and diabetes insipidus. Anterior pituitary deficiency is relatively rare and, although hypothalamic involvement with LCH is common (25), precocious puberty is extremely rare. As a generalization, children with LCH do not have anterior pituitary failure unless they have diabetes insipidus (25).
http://www.endotext.org/neuroendo/neuro ... ndo11a.htm
Approximately 90% of vasopressin secretion needs to be interrupted before there is clinical diabetes insipidus. The commonest tumors causing diabetes insipidus in childhood are craniopharyngioma and germinoma. The latter are often occult for many years and patients require serial neuro-imaging before the diagnosis may be revealed. It may be as long as 21 years from the onset of diabetes insipidus to imaging the tumor (22). In a child who has diabetes insipidus, it is important to monitor carefully growth and screen for an evolving anterior pituitary endocrinopathy.
Lesions in the pituitary stalk associated with the presence of diabetes insipidus, are usually situated in the middle of the stalk and related to a dural inflammatory process, such as sarcoidosis or Langerhans cell histiocytosis (LCH) (23). Langerhans cell histiocytosis is not a cancer, but has similar characteristics and is a clonal proliferation of abnormal histiocytes, the Langerhans cells (24). A common presentation of LCH in the middle childhood years, which used to be described as Hand-Schüller-Christian disease, are small punched-out skull lesions associated with pituitary stalk thickening and diabetes insipidus. Anterior pituitary deficiency is relatively rare and, although hypothalamic involvement with LCH is common (25), precocious puberty is extremely rare. As a generalization, children with LCH do not have anterior pituitary failure unless they have diabetes insipidus (25).
http://www.endotext.org/neuroendo/neuro ... ndo11a.htm
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Melnichenko
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- Откуда: москва